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Treinamento muscular inspiratorio na doenca pulmonar obstrutiva cronica: impacto na qualidade de vida, intolerancia ao esforco e dispneia (Inspiratory muscle training in chronic obstructive pulmonary disease: impact on quality of life, exercise intolerance, and dyspnea) [Portuguese]
di Mambro TR, Figueiredo PHS, Wanderley TR, Kristki AL, Guimaraes FS
Fisioterapia e Pesquisa [Physical Therapy and Research] 2007 May-Aug;14(2):65-71
clinical trial
5/10 [Eligibility criteria: Yes; Random allocation: Yes; Concealed allocation: No; Baseline comparability: Yes; Blind subjects: No; Blind therapists: No; Blind assessors: Yes; Adequate follow-up: No; Intention-to-treat analysis: No; Between-group comparisons: Yes; Point estimates and variability: Yes. Note: Eligibility criteria item does not contribute to total score] *This score has been confirmed*

Inspiratory muscle training (IMT) has shown beneficial effects concerning inspiratory muscle function, but its effects on exercise intolerance and quality of life are not clearly established. The aim of this study was to evaluate IMT effects in patients with mild to severe chronic obstructive pulmonary disease (COPD), concerning respiratory muscle strength, quality of life (as measured by the SF-36 questionnaire), exercise intolerance (measured by the 6MWD, six-minute walking distance test), and dyspnea (measured by the MMRC Modified Medical Research Council Scale). Twenty-five patients were divided into training (TG, n = 12) and control (CG, n = 13) groups. IMT protocol lasted 20 minutes, 5 days a week during 6 weeks, with a workload of 40% of maximal inspiratory pressure (Pimax). Results showed a higher increase of Pimax in TG (52 +/- 6 to 82 +/- 7 cmH2O) as compared to CG (58 +/- 4 to 60 +/- 6 cmH2O; p < 0.05). Dyspnea improved in eight TG patients and did not change in CG (p < 0.05). No statistically significant changes were found in 6MWD test data or SF-36 scores, although all TG questionnaire scores were higher at the final evaluation. We conclude that the IMT protocol used increases respiratory muscle strength and reduces dyspnea in mild to severe COPD patients.

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